Fig. 10From: Role of ATF3 triggering M2 macrophage polarization to protect against the inflammatory injury of sepsis through ILF3/NEAT1 axisSchematic drawing of ATF3/ILF3/NEAT1 axis in M2 polarization. ATF3 transcriptionally suppresses ILF3 expression, thus impairing the stability of NEAT1, ultimately promoting M2 polarizationBack to article page