Correction: Angiotensin-Converting Enzyme (ACE) 2 Overexpression Ameliorates Glomerular Injury in a Rat Model of Diabetic Nephropathy: A Comparison with ACE Inhibition

Correction: Molecular Medicine 17, (2011) 59-69 https://0-doi-org.brum.beds.ac.uk/10.2119/molmed.2010.00111

Following publication of the original article [1], the authors found an error in Fig. 1B: the immunohistochemical images in the ACEI and AD-ACE2 + ACEI groups were mistakenly used, which were partially overlapped with the correct image in the Ad-ACE2 group. They went back to their raw data and found the original immunohistochemical images in the ACEI and AD-ACE2 + ACEI groups (n = 10 in each group). The corrected Fig. 1B is given in this correction article.

Fig. 1

Efficiency of gene transfer 4 wks after gene delivery. A Fluorescence microscopic images of frozen sections of the kidneys in the normal, no treatment, Ad-GFP, Ad-ACE2, ACE1 and Ad-ACE2 + ACEI groups, respectively (bar = 50 μm). G indicates glomeruli and T indicates tubules. B Immunochemical staining showing the localization of ACE2 protein in cortical sections from each group. Weak ACE2 staining was observed in tubules from rats in the no treatment and Ad-GFP groups, whereas marked ACE2 staining was depicted in both tubules and glomeruli from rats in the normal, Ad-ACE2, ACEI and Ad-ACE2 + ACEI groups (bar = 50 μm). C, D, E Quantitative analysis of ACE2 mRNA levels, protein levels and activities. *P < 0.05, ^#P < 0.01 versus normal group; ^‡P < 0.05, ^§P < 0.01 versus no treatment group

The authors apologise for this error.